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Still, if they don’t sign him to a long-term deal before July 16, the Steelers may end up having to find a way to thrive without Bell in 2019, and beyond.

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Absurd amounts of money paid to these people. Main reason so many fans can’t afford to attend games. Soon the stands will be filled with corporate types who only want to tell their friends that they attended a game, even if they weren’t sure who was playing.

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. The Steelers took S Terrell Edmunds at pick #28. The Patriots took RB Sony Michel at #31. One could play the “what if?” game as in, ” What if the Steelers cut Bell and beat the Patriots to Michel?” Only time will tell which was the better move. . .

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There are 2 components to a players worth: skill and price

Tom Brady is a bargain at $20 million/year. He would be a terrible signing at $100 million/year.

LeVeon Bell is a great player skill wise. At 12 million he’s worth having. Is he worth having at 15MM? 18MM? At some point the team is paying so much that regardless of how good the player is, he’s costing the team too much. They have to let 2 other good players go and replace them with scrubs.

Bell should sign the contract, take his money, and then make more for the next couple of years.

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The rams are in serious trouble in their very tight window.

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bocadiver1 says: “Absurd amounts of money paid to these people. Main reason so many fans can’t afford to attend games.” ================================

Bull. The bulk of the NFL revenue is from tv contracts. Obviously, you’ve never been to a real NFL game, as I only see fans everywhere around us.

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The classic mechanism of ST-elevation MI is complete occlusion (typically thrombosis or embolism) of a coronary artery. In contrast, NSTEMI is usually a result of a transient or near-complete occlusion of a coronary artery or acute factor that deprives myocardium of oxygen.

Unstable plaques have soft, lipid-laden contents, with thin, often sclerotic fibrous caps infiltrated by macrophages (foam cells). Release of the lipid-rich atherogenic core causes adhesion, activation, and aggregation of platelets. This initiates the coagulation cascade. A superimposed thrombus forms, occluding the coronary blood flow and resulting in myocardial ischemia causing a type 1 MI.

NSTEMI may also be caused by other mechanisms, such as dynamic obstruction (i.e., focal coronary artery spasm or Prinzmetal angina), severe progressive atherosclerosis, restenosis following percutaneous coronary intervention (PCI), recreational drug use (e.g., cocaine or other stimulants), arterial inflammation, or extrinsic causes leading to myocardial supply-demand mismatch (i.e., type 2 MI precipitated by acute blood loss in a patient with underlying CAD).

NSTEMI is a result of an acute imbalance between myocardial oxygen demand and supply, most commonly due to a reduction in myocardial perfusion. Type 1 MI is most commonly caused by a nonocclusive thrombus that develops in a disrupted atherosclerotic plaque, and leads to nonocclusive or near-complete thrombosis of a vessel supplying the myocardium.

Several different sequences of events may lead to an NSTEMI:

The most common cause is plaque rupture or obstructive atherosclerotic disease. In this setting, the release of myocardial biomarkers in type 1 MI is thought to be due to atherosclerotic plaque fissuring or rupture with resulting intracoronary thrombus or platelet emboli leading to diminished myocardial blood flow.

Plaque rupture usually occurs at the weakest and thinnest part of the atherosclerotic cap (often at the shoulder region). Ruptured plaques contain large numbers of inflammatory cells including monocytes, macrophages, and T lymphocytes. Although one third of occlusions occur at a site with the greatest stenosis, most (66% to 78%) arise from lesions with <50% stenosis, and <5% arise from lesions exhibiting >70% stenosis.

It is thought that the lack of ST elevation is because the infarct does not involve the full thickness of the myocardium (not a transmural infarction).

The severity of myocardial damage in NSTEMI depends on:

Classically it is thought that NSTEMI patients ultimately have a diagnosis of a non-Q-wave MI; however, 25% of patients with NSTEMI and elevated biomarkers go on to develop Q-wave MI in the weeks to follow. In addition, approximately 25% of patients with a diagnosis of NSTEMI have a 100% occlusion of the affected artery on coronary angiography.

Plaque rupture with superimposed nonocclusive thrombus or embolic events leading to coronary vascular obstruction

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Congress established the Food Stamp Program in 1964 to “safeguard the health and well-being of the Nation’s population by raising levels of nutrition among low-income households.” 1 Now known as the Supplemental Nutrition Assistance Program, SNAP is a federally funded and state-administered program that “permit[s] low-income households to obtain a more nutritious diet through normal channels of trade by increasing food purchasing power for all eligible households who apply for participation.” 2 The U.S. Food and Nutrition Service is the federal agency in charge of SNAP.

While the Personal Responsibility and Work Opportunity Reconciliation Act of 1996 was primarily aimed at changing federal cash assistance, it put restrictions on other public benefits including SNAP. 3 Among these restrictions was a little-noticed provision that barred unemployed adults aged 18–49 who are not disabled or raising minor children from receiving SNAP for more than three months in any three-year period. 4 These affected individuals are referred to as Able-Bodied Adults Without Dependents, or ABAWDs.

The federal government suspended the ABAWD time limit for 2009 and 2010 as part of the American Recovery and Reinvestment Act. 5 After 2010 many states qualified for (and the U.S. Food and Nutrition Service approved) statewide waivers of the time limit in light of persistently high unemployment. As employment conditions improved over the last few years, states are once again required by federal law to implement the ABAWD time limit. More than 40 states implemented the time limit in 2016 in at least some areas in their states, and 22 of those states implemented the time limit for the first time since the Great Recession. 6 One study estimated that anywhere from 500,000 to one million people would lose SNAP benefits in 2016 because of the reimposition of the time limit, and, in fact, April 2016 saw a 773,000-person drop in SNAP participation driven by the time limit. 7 Currently 26 states have partial waivers from the ABAWD time limit; 17 have implemented the ABAWD time limit statewide; and 7 states and Washington, D.C., Guam, and the U.S. Virgin Islands have statewide waivers. 8 As a result, ensuring lawful implementation of the ABAWD time limit is a pressing issue to both state agencies and civil legal aid lawyers nationwide.

The SNAP time limit does not apply to all SNAP recipients. Six categories of recipients are exempt from the time limit: (1) those under 18 or 50 years of age or older, (2) those medically certified as physically or mentally unfit for employment, (3) parents of a household member under 18, even if the household member who is under 18 is not eligible for SNAP, (4) those who reside in a household where a household member is under 18, even if the household member who is under 18 is not eligible for SNAP, (5) those who are otherwise exempt from work requirements under section 6(d)(2) of the Food Stamp Act, and (6) those who are pregnant. 9

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